Articles – D.R.A.S.T.I.C. Research

How an Indian scientist couple worked to trace origin, course of COVID-19

By Dr Monali C. Rahalkar And Dr Rahul BahulikarJune 13, 2021 16:47 IST

BSL Biological Laboratories and Coronaviruses in Wuhan, China — Short Description

a DRASTIC study

Prometheus & Pandora I: Trust the Scientists on COVID, not the Science?

Paved with good intentions.

Prometheus & Pandora II: The West must not go gently into a COVID-19 goodnight

The value of peace must never exceed that of freedom or justice.

Prometheus & Pandora III: The Apocryphal Origin of SARS-CoV-2

The ‘Consensus’ of scientists supporting a zoonotic origin for COVID-19 is a false premise

Learning from the past – or not.

Anybody who really wants to understand China’s attitude today would benefit from reading Chapter 5 of this excellent book: bit.ly/3fzHFbr

White Unicorns – or the silly stories about no Covid-19 positive tests at the WIV (and other labs)

Issue #1:Sorry @China__Focus, but you have been caught lying.Ignoring your 2019 typo (should be 2020, the 700 tests ILI samples from the Wuhan Xiehe Hospital – not from the WIV (which anyway has 590 staff and students).

We need to talk about the ToRs

Here we examine the circumstances behind the redaction of the Terms-of-Reference (ToRs) of the Global Study of the Origins of SARS-COV2.

WHO knows? We still can’t be sure of Covid’s origins

Could Covid-19 have originated from a Chinese lab accident? When the virus was first identified, many pointed out that it was close to the Wuhan Institute of Virology. It sounded like a conspiracy theory. But it’s all too plausible. And there are questions that the long-awaited report by the World Health Organisation leaves unanswered.

The RaTg13 S does not bind to any ACE2 with affinity and entry capacity that is viable for Sarbecoviruses.

The RaTg13 S does not bind to any ACE2 with affinity and entry capacity that is viable for Sarbecoviruses. This includes both human ACE2 and ACE2 Ra4331, which had even lower binding efficiency and infection efficiency than human ACE2. (Click on image for large view) The SARS-CoV-2 S is uniquely adapted to human ACE2. Reference:Continue reading “The RaTg13 S does not bind to any ACE2 with affinity and entry capacity that is viable for Sarbecoviruses.”